Wednesday, March 19, 2025

GASTROINTESTINAL SYSTEM

 

 GASTROINTESTINAL SYSTEM



1. ANATOMY OF THE GASTROINTESTINAL SYSTEM

1.1 General Features of the GI System

Derived from endoderm (mucosa) and mesoderm (muscle, serosa, blood supply).
Major Functions:

  • Digestion → Breakdown of food into absorbable molecules.
  • Absorption → Uptake of nutrients into the bloodstream.
  • Secretion → Enzymes, mucus, and hormones for digestion and motility.
  • Excretion → Removal of waste products via feces.
  • Immune Function → Gut-associated lymphoid tissue (GALT).

1.2 Divisions of the GI System

Divided into Upper and Lower GI Tract based on anatomical and functional roles.

1.2.1 Upper GI Tract

Mouth (Oral Cavity) → Mastication, saliva production, taste perception.
Pharynx → Common pathway for food and air, contains tonsils for immune defense.
Esophagus → Muscular tube with upper and lower esophageal sphincters (UES, LES).
Stomach → Acidic environment for protein digestion, churns food into chyme.

1.2.2 Lower GI Tract

Small Intestine → Primary site for digestion and absorption.
Large Intestine (Colon) → Water absorption, stool formation, gut microbiota.
Rectum & Anus → Waste elimination, controlled by internal and external anal sphincters.


1.3 Layers of the GI Tract (From Inner to Outer)

Mucosa → Epithelium, lamina propria (GALT), muscularis mucosa.
Submucosa → Dense connective tissue, Meissner’s plexus (secretion control).
Muscularis Externa → Inner circular and outer longitudinal muscles, Auerbach’s plexus (motility).
Serosa (Adventitia in Some Areas) → Outermost protective layer, peritoneal covering.


1.4 Accessory Organs of Digestion

Salivary Glands → Parotid, submandibular, sublingual (produce enzymes like amylase).
Liver → Bile production, detoxification, glycogen storage, protein synthesis.
Gallbladder → Stores bile, releases it via common bile duct.
Pancreas → Exocrine (enzymes like lipase, amylase) & endocrine (insulin, glucagon) functions.


2. PHYSIOLOGY OF THE GASTROINTESTINAL SYSTEM

2.1 Regulation of GI Motility

✔ Controlled by Enteric Nervous System (ENS) and Autonomic Nervous System (ANS).
Myenteric (Auerbach’s) Plexus → Controls peristalsis & motility.
Submucosal (Meissner’s) Plexus → Regulates secretion & blood flow.
Key Movements:

  • Peristalsis → Propels food forward.
  • Segmentation → Mixing movements for digestion.
  • Mass Movements → Large bowel contractions before defecation.

2.2 Digestion & Absorption

Carbohydrates → Digested by amylase, absorbed as monosaccharides.
Proteins → Pepsin & pancreatic proteases break them into amino acids.
Fats → Emulsified by bile, digested by lipases into fatty acids.
Vitamins & Minerals:

  • Vitamin B12 → Absorbed in the ileum via intrinsic factor.
  • Iron → Absorbed in the duodenum.

2.3 Hormonal Regulation of Digestion

Gastrin → Stimulates acid secretion in the stomach.
Cholecystokinin (CCK) → Stimulates gallbladder contraction & pancreatic secretion.
Secretin → Stimulates bicarbonate secretion from pancreas.
Ghrelin → Increases appetite.
Leptin → Decreases appetite.


3. PATHOLOGY OF THE GASTROINTESTINAL SYSTEM

3.1 Esophageal Disorders

Gastroesophageal Reflux Disease (GERD) → Acid reflux due to LES dysfunction.
Achalasia → Failure of LES relaxation, "bird’s beak" sign on barium swallow.
Esophageal Cancer → Squamous cell carcinoma (upper) vs. Adenocarcinoma (lower).


3.2 Stomach Disorders

Gastritis → Inflammation due to H. pylori or NSAIDs.
Peptic Ulcer Disease (PUD) → Duodenal (pain relieved by food) vs. Gastric ulcers.
Gastric Cancer → Associated with H. pylori, smoking, nitrates.


3.3 Intestinal Disorders

Malabsorption Syndromes:

  • Celiac Disease → Gluten intolerance, villous atrophy.
  • Lactose Intolerance → Deficiency of lactase enzyme.
    Inflammatory Bowel Disease (IBD):
  • Crohn’s Disease → Skip lesions, transmural inflammation, granulomas.
  • Ulcerative Colitis → Continuous lesions, mucosal inflammation, crypt abscesses.
    Colorectal Cancer → Right-sided (bleeding), left-sided (obstruction).

3.4 Hepatobiliary & Pancreatic Disorders

Hepatitis → Viral (A, B, C, D, E), autoimmune, alcoholic.
Cirrhosis → Liver fibrosis, portal hypertension, ascites.
Cholelithiasis (Gallstones) → Cholesterol (most common) vs. Pigment stones.
Pancreatitis → Acute (enzymatic autodigestion) vs. Chronic (irreversible damage).


4. CLINICAL EXAMINATION OF THE GASTROINTESTINAL SYSTEM

4.1 History Taking

Dysphagia → Difficulty swallowing (oropharyngeal vs. esophageal).
Abdominal Pain → Localized vs. generalized (visceral or somatic).
Altered Bowel Habits → Diarrhea, constipation, steatorrhea.
GI Bleeding → Hematemesis (upper GI), melena (black stool), hematochezia (fresh blood in stool).


4.2 Physical Examination

Inspection → Jaundice, distension, scars, striae, caput medusae.
Palpation → Organomegaly (liver, spleen), tenderness (McBurney’s point for appendicitis).
Percussion → Shifting dullness (ascites), liver span measurement.
Auscultation → Bowel sounds (hyperactive in obstruction, absent in ileus).


4.3 Special Tests

Murphy’s Sign → Positive in acute cholecystitis.
Rovsing’s Sign → Positive in appendicitis.
Psoas Sign → Indicates retrocecal appendicitis.
Courvoisier’s Sign → Painless jaundice + enlarged gallbladder (pancreatic cancer).


5. HIGH-YIELD PG EXAM PEARLS

GERD Treatment → PPI > H2 blockers > Lifestyle changes.
Crohn’s vs. UC → Cobblestone (Crohn’s) vs. Lead-pipe (UC) appearance.
Acute Pancreatitis Causes → Gallstones, Alcohol (most common).
Cirrhosis Marker → AST > ALT (alcoholic liver disease).



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